THE AIDS INDUSTRY AND MEDIA WANT YOU TO THINK THERE ARE ONLY A HANDFUL OF SCIENTISTS WHO DOUBT THE HIVAIDS THEORY.
HERES THE REALITY.
Release of membrane vesicles, a process conserved in both prokaryotes and eukaryotes, represents an evolutionary link, and suggests essential functions of a dynamic extracellular vesicular compartment (including exosomes, microparticles or microvesicles and apoptotic bodies). Compelling evidence supports the significance of this compartment in a broad range of physiological and pathological processes. However, classification of membrane vesicles, protocols of their isolation and detection, molecular details of vesicular release, clearance and biological functions are still under intense investigation.György B et al. Membrane vesicles, current state-of-the-art: emerging role of extracellular vesicles. Cell Mol Life Sci. 2011 Aug;68(16):2667-88.In recent years a wide range of regulatory functions have been attributed to membrane vesicles with the hallmarks of exosomes [virus-like particles originating from a cell]. The identification of vesicles as exosomes is based on both morphological and biochemical criteria although particular attention should be given to the purification and characterization methods. Exosomes are the ILVs of MVBs and are thus endosome-derived membrane vesicles. An exosomal-type of communication is certainly not limited to the immune system and may be exploited by cells to interact with each other, locally or at a distance. Highlighting the cellular and molecular basis of exosome targeting to recipient cells will be invaluable to understand how different populations of exosomes maintain their own specificity. Exosome biogenesis is still far from being well understood and the mechanisms involved in the fusion of secretory MVBs with the plasma membrane are unknown.van Niel G et al. Exosomes: a common pathway for a specialized function. J Biochem. 2006 Jul;140(1):13-21.Retroviruses are enveloped viruses that are generally assumed to bud at the plasma membrane of infected cells. Recently it has become apparent that some of these viruses use the endocytic pathway to coordinate their assembly and release. In addition, these and some other enveloped viruses exploit the machinery that generates the internal membranes of multivesicular bodies (MVB). These observations and others have led to the suggestion that retroviruses be regarded as viiral exosomes. [in other words, retroviruses are virtually identical to cell-derived exosomes an, who knows, could actually be completely cellular, and not infectious, entities]Pelchen-Matthews A, Raposo G, Marsh M. Endosomes, exosomes and Trojan viruses. Trends Microbiol. 2004 Jul;12(7):310-6.Human immunodeficiency virus types 1 and 2: (E. coli, B. megaterium, recombinant antigen) HIVAB HIV-1/HIV-2 (rDNA) EIA. Abbott Laboratories. 2004
http://davidcrowe.ca/SciHealthEnv/papers/5017-Abbott-EIA.pdfLipid rafts are specialized regions of cell membranes enriched in cholesterol and sphingolipids that are involved in immune activation and signaling. Studies in T-cells indicate that these membrane domains serve as sites for release of human immunodeficiency virus (HIV). By budding through lipid rafts in T-cells, HIV selectively incorporates raft markers and excludes non-raft proteins our data demonstrate discordance in the abundance of some proteins in lipid rafts and in HIV. Finally, our data reveal a strong concordance between the host cell protein profile of exosomes and that of HIV. These results strongly support the Trojan exosome hypothesis and its prediction that retroviral budding represents exploitation of a pre-existing cellular pathway of intercellular vesicle trafficking. [and that cellular exosomes are very similar to what are called retroviruses]Nguyen DG et al. Evidence that HIV budding in primary macrophages occurs through the exosome release pathway. J Biol Chem. 2003 Dec 26;278(52):52347-54.During preliminary experiments to establish the proportion of virus-coded p24 protein to virus membrane-associated HLA-DR in gradient-enriched HIV-1 preparations, we became aware of a large variability between experiments. In order to determine whether HLA-DR-containing cellular material was contaminating the virus preparations, we carried out enrichment by gradient centrifugation of clarified supernatants from noninfected cells and tested this material for HLA-DR content. We found that, independently of the cell type used, gradient enrichment resulted in the isolation of large quantities of HLA-DR-containing material which banded at a density overlapping that of infectious HIV. Electron microscopy of gradient-enriched preparations from supernatants of virus-infected cells revealed an excess of vesicles with a size range of about 50-500 nm, as opposed to a minor population of virus particles of about 100 nm. Electron micrographs of infected cells showed polarized vesiculation of the cell membrane, and virus budding was frequently colocalized with nonviral membrane vesiculation. Analysis of the cellular molecules present in the fractions containing virus or exclusively cellular material demonstrated that virus and cellular vesicles share several cellular antigens, with the exception of CD43 and CD63, found mainly at the virus surface, and HLA-DQ, which was found only in the cellular vesicles.Gluschankof P et al. Cell membrane vesicles are a major contaminant of gradient-enriched human immunodeficiency virus type-1 preparations. Virology. 1997 Mar 31;230(1):125-133.The third type of smaller particle [found in cell cultures using a negative staining Transmission Electron Microscope], with surface spikes, was found The small particles were present alongside immature and mature HIV particles but they were less numerous than HIV. The particles were usually spherical but sometimes slightly angular in shape and 65 to 90 nm in diameter [too small to be HIV] The small spiked particles were not detected by negative staining in a preparation made from a control CEM culture not infected with HIV [so, they are not HIV, but are commonly associated with HIV. What are they? How is it known that some HIV proteins do not belong to these particles? How is it known that these are not viruses, but that the larger particles are?]Hockley DJ et al. Electron microscopy of human immunodeficiency virus. J Gen Virol. 1988 Oct;69 (Pt 10):2455-69.We have examined HIV-1 and HIV-2 infected cell cultures by negative staining and IEM [Immune Electron Microscopy], using a range of clinical and monocolonal antibodies. The study was complicated by the presence of a viruslike structure 70-80 nm in diameter, too small to be HIV HIV-2 cultures also contained large numbers of 130-300-nm particles [too large to be HIV]. The majority of these particles had no detectable fringe and when penetrated by stain were frequently seen to contain an elongated internal core Subsequent careful examination of the H9/RF (HIV-1) cell line revealed particles having the same morphology [structure] only at a level approaching the sensitivity of the techniqueChrystie IL, Almeida JD. The morphology of human immunodeficiency virus (HIV) by negative staining. J Med Virol. 1988 Jul;25(3):281-8.Lymph nodes from the PGL [persistent generalized lymphadenopathy - swollen lymph nodes] group [of people believed at risk of AIDS] were characteristically enlarged In 11 of these 18 lymph nodes, occasional particles were identified that measured approximately 100 nm in diameter with well-delineated outer limiting membranes surrounding central to eccentric predominantly round electron-dense cores [i.e. the size, shape and structure of HIV] [however] in 7 of 18 no cored particles were identified despite an intensive search. In two of the 20 PGL lymph nodes neither type of particle [with or without cores] were found [we also studied] non-HIV related reactive lymph nodes [although all the people from which samples were taken were assumed to be HIV-negative, none were actually tested]. Of major significance was the finding of viral-like particles, morphologically [i.e. size, shape and structure] indistinguishable from those observed in PGL cored particles were seen in 8 of the 15 lymph nodes The results of this study compel us to conclude that, while the particles observed in PGL lymph nodes are indeed HIV [because 19/20 had anti-p24 staining, while none of the non-HIV lymph nodes did], morphologically similar particles can be seen in other reactive conditions, and the presence of such particles do not, by themselves, indicated infection by HIV. Clearly, techniques that demonstrate the presence of specific viral antigens or viral RNA are needed to supplement ultrastructural observations [or even actual isolation of the putative virus]O'Hara CJ et al. The ultrastructural and immunohistochemical demonstration of viral particles in lymph nodes from human immunodeficiency virus-related and non-human immunodeficiency virus-related lymphadenopathy syndromes. Hum Pathol. 1988 May;19(5):545-9.We used the characteristic cylindrical structure in the core [of the supposed virus particle] as an identifying characteristic for the virus to distinguish it from cellular debris and also noted that it may vary considerably in its dimensions and morphological features We have found two basic virus particle sizes, 90 nm and 120 nm, both present in large numbers. The larger particle bears no surface projections, while the smaller particle is rarely 'naked' and usually bears projections the existence of small, mature particles with projections remains unexplained [as does the presence of at least two different sizes when a virus should be very consistent in size]Lecatsas G et al. Pleomorphism in HTLV-III, the AIDS virus. S Afr Med J. 1986 Jun 21;69(13):793-4.
There you have it. No handful of wild-eyed conspiracy theorists. No right-wing racists, as the Aids industrys spinmeisters would have you believe. Just 19 very serious, concerned, highly educated people from every corner of the globe who sense that an enormous tragedy is unfolding due to the medical establishments unwillingness to face the evidence that the Hiv-Aids theory is a mistake.
The people on this page were intellectually curious enough to have sought out and studied the arguments that discredit the Hiv-Aids theory. Since the mass media and professional journals censor these arguments, the vast majority of doctors and scientists, although decent people who want to do the right thing, have never been exposed to them, and so accept the biased conclusions of politicized bureaucracies like the CDC and WHO, whose coziness with the drug industry is legendary and whose recommendations always seems to dovetail perfectly with drug industry marketing plans.
Were it not for the massive media blackout of information that contradicts the Hiv theory, many more people would be asking tough questions.
The next time you hear the media say, only a handful of scientists doubt Hivs role in Aids, refer them to this page. Explain to them that it is wrong to misrepresent the fact that there is enormous dissent to the Hiv-Aids paradigm.
The next time you hear the media drone, Hiv, the virus that causes Aids, remind them that journalists are supposed to distinguish between what is a theory and what is a fact. That Hiv-Aids is only a theory and has never been proven, is admitted by top scientists even in the Aids establishment.
The next time the media announce that tens of millions of people are dying from Hiv in Africa, ask them how they know that. Remind them that journalists are supposed to question dubious assertions from powerful, drug-industry funded agencies like the WHO, not parrot them as if they were indisputable. Ask them why they report these numbers as if they were actual Aids cases, when in fact they are projections made by WHOs computer programs, based on very questionable statistical methodologies and contradicted by many facts including the continual large population increases experienced in the countries supposedly worst affected.
Request that the media stop twisting the truth in support of a politicized, entrenched Aids establishment that profits financially by terrorizing people, pokes its nose shamelessly into peoples private sex lives, compels people to submit to inaccurate tests and literally forces mothers and babies to swallow toxic, unproven chemotherapy drugs with horrific, often-fatal side effects.
Explain to them that this is irresponsible, and that such actions cause needless anxiety, shatter peoples lives, tear families apart, destroy hope and trigger countless suicides. And that while we realize that sensational headlines about killer viruses sell newspapers, the social cost of these profits is unacceptable.
Make the media understand that keeping people in the dark about the large number of credentialed dissenters to the Hiv-Aids dogmas, and the financial conflicts of interest that are rampant among Hiv-Aids scientists and NGOs, is a violation of everyones human right to informed consent and freedom of information.
Note: Affiliation with an organization does not imply that the organization supports the individual HIV/AIDS skeptics position.
Does Hiv cause Aids? Lots of scientists say no. Read more.
Alive And Well
Dr. Peter Duesberg
The Perth Group
Treatment Information Group
Immunity Resource Foundation
Alberta Reappraising Aids Society
Last updated January 29, 2013.